Get 10 ½ Things No Commencement Speaker Has Ever Said PDF

By Charles Wheelan

ISBN-10: 0393084574

ISBN-13: 9780393084573

Modeled on Charles Wheelan’s 2011 category Day Speech at Dartmouth collage, this choice of refreshingly sincere recommendation and observations is the antidote to these cotton-candy platitudes which are all too widely used to somebody who’s ever worn a mortarboard. Armed with a PhD in public coverage, a long time of expertise in social technology learn, and—perhaps so much important—good-natured humor, Wheelan bargains up 10½ head-turning aphorisms on happiness and luck that any one staring down the barrel of commencement must pay attention yet most likely hasn’t heard but. Celebrated New Yorker cartoonist Peter Steiner provides a marginally of caprice along with his irreverent illustrations sprinkled all through.

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Extra resources for 10 ½ Things No Commencement Speaker Has Ever Said

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The cell is smaller in size; the cytoplasm is dense (Fig. 1–22A); and the organelles, A though relatively normal, are more tightly packed. ) Chromatin condensation. This is the most characteristic feature of apoptosis. The chromatin aggregates peripherally, under the nuclear membrane, into dense masses of various shapes and sizes (Fig. 1–22B). The nucleus itself may break up, producing two or more fragments. Formation of cytoplasmic blebs and apoptotic bodies. The apoptotic cell first shows extensive surface blebbing, then undergoes fragmentation into membrane-bound apoptotic bodies composed of cytoplasm and tightly packed organelles, with or without nuclear fragments (Fig.

Decreased phospholipid synthesis. The production of phospholipids in cells may be reduced as a consequence of Cytosolic Ca2+ O2 Reactive oxygen species Lipid peroxidation ATP Phospholipase activation Protease activation Phospholipid degradation Cytoskeletal damage Phospholipid reacylation/ synthesis Phospholipid loss Lipid breakdown products MEMBRANE DAMAGE FIGURE 1–21 Mechanisms of membrane damage in cell injury. Decreased O2 and increased cytosolic Ca2+ are typically seen in ischemia but may accompany other forms of cell injury.

This reduces the intracellular pH, resulting in decreased activity of many cellular enzymes. Failure of the Ca2+ pump leads to influx of Ca2+, with damaging effects on numerous cellular components, described below. With prolonged or worsening depletion of ATP, structural disruption of the protein synthetic apparatus occurs, manifested as detachment of ribosomes from the rough ER and dissociation of polysomes, with a consequent reduction in protein synthesis. In cells deprived of oxygen or glucose, proteins may become misfolded, and misfolded proteins trigger a cellular reaction called the unfolded protein response that may culminate in cell injury and even death.

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10 ½ Things No Commencement Speaker Has Ever Said by Charles Wheelan


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